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0.39 /METABOLISMO . 
0.17 CANDIDA .
0.17 BOCA .
0.17 /QUIMICA .
0.10 ALDEIDO DESIDROGENASE .

Rank Sim. Fonte Título, abstract e conceitos DeCS   Conceitos DeCS em LILACS 
1 0.59 LILACS Increased salivary aldehyde dehydrogenase 1 in non-reticular oral lichen planus^ien . 
ehydrogenase may be one of the malignant transformation markers in oral lichen planus Further studies are needed for introducing aldehyde dehydrogenase as a prognostic indicator in certain lesions^ien] + Increased salivary aldehyde dehydrogenase 1 in non-reticular oral lichen planus^ien; An bras dermatol .
Abstract: Background: Oral lichen planus is a potentially malignant disorder One of the malignant transformation markers is cancer stem cells One of the proposed marker for the detection of cancer stem cells's in head and neck cancer is aldehyde dehydrogenase Recently it is shown that aldehyde dehydrogenase 1 expression in tissue samples is associated with oral lichen planus malignant transformat .. ehydrogenase may be one of the malignant transformation markers in oral lichen planus Further studies are needed for introducing aldehyde dehydrogenase as a prognostic indicator in certain lesions^ien .
  0.20 /PATOLOGIA . 
0.20 NEOPLASIAS BUCAIS .
0.20 LIQUEN PLANO BUCAL .
0.20 CANDIDA .
0.19 BOCA .
2 0.43 LILACS Expression of aldehyde dehydrogenase after neoadjuvant chemotherapy is associated with expression of hypoxia-inducible factors 1 and 2 alpha and predicts prognosis in locally advanced breast cancer^ien . 
[OBJECTIVE: To analyze the expression of hypoxia-inducible factors (hypoxia-inducible factor 1A and hypoxia-inducible factor 2A) and aldehyde dehydrogenase proteins in patients with locally advanced breast carcinoma who were subjected to neoadjuvant chemotherapy METHODS: We included 90 patients with histologically confirmed stage II and III breast carcinoma who were treated with neoadjuvant chemoth .
OBJECTIVE: To analyze the expression of hypoxia-inducible factors (hypoxia-inducible factor 1A and hypoxia-inducible factor 2A) and aldehyde dehydrogenase proteins in patients with locally advanced breast carcinoma who were subjected to neoadjuvant chemotherapy METHODS: We included 90 patients with histologically confirmed stage II and III breast carcinoma who were treated with neoadjuvant chemoth .. educed overall survival CONCLUSION: The presence of aldehyde dehydrogenase-positive cells within the residual tumor after neoadjuvant chemotherapy is associated with an increase in the expression ^ien .

  0.62 /METABOLISMO . 
0.15 NEOPLASIAS DA MAMA .
0.09 FATOR 1 INDUZIVEL POR HIPOXIA .
0.09 ALDEIDO DESIDROGENASE .
0.06 FATORES DE TRANSCRICAO HELICE-ALCA-HELICE BASICOS .
3 0.40 LILACS Sperm especific lactate dehydrogenase^ien . 

  0.65 L-LACTATO DESIDROGENASE . 
0.16 ESPERMATOZOIDES .
0.06 LACTATO DESIDROGENASES .
0.06 ACIDO OXAMICO .
0.06 ELECTROPHORUS .
4 0.39 LILACS Evaluation of different dehydrogenases to recognize Candida species commonly isolated from human oral cavities^ien . 
[Electrophoresis of some dehydrogenases were carried out in order to establish relatedness degrees among five Candida species commonly isolated from oral cavity of humans, by numerical taxonomy methods The obtained data revealed that some dehydrogenases are capable of distinguishing strains of different species, but most of these enzymes could not organize all strains in their respective clusters N .
Electrophoresis of some dehydrogenases were carried out in order to establish relatedness degrees among five Candida species commonly isolated from oral cavity of humans, by numerical taxonomy methods The obtained data revealed that some dehydrogenases are capable of distinguishing strains of different species, but most of these enzymes could not organize all strains in their respective clusters N .. clusters Numerical classifications based on dehydrogenase polymorphism must be considered for surveys involving just one species of yeast genus, where this resource had already shown to be useful^ien .

  0.40 CANDIDA . 
0.25 BOCA .
0.15 OXIRREDUTASES .
0.10 /ISOLAMENTO & PURIFICACAO .
0.10 /MICROBIOLOGIA .
5 0.39 LILACS Relationship between acetaldehyde concentration in mouth air and tongue coating volume^ien . 
[Objective Acetaldehyde is the first metabolite of ethanol and is produced in the epithelium by mucosal ALDH, while higher levels are derived from microbial oxidation of ethanol by oral microflora such as Candida species However, it is uncertain whether acetaldehyde concentration in human breath is related to oral condition or local production of acetaldehyde by oral microflora The aim of this pilo .
Objective Acetaldehyde is the first metabolite of ethanol and is produced in the epithelium by mucosal ALDH, while higher levels are derived from microbial oxidation of ethanol by oral microflora such as Candida species However, it is uncertain whether acetaldehyde concentration in human breath is related to oral condition or local production of acetaldehyde by oral microflora The aim of this pilo .. clinical parameters, presence of Candida species, smoking status or alcohol sensitivity Conclusion Physiological acetaldehyde concentration in mouth air was associated with tongue coating volume ^ien .

  0.36 /QUIMICA . 
0.19 /ANALISE .
0.19 /METABOLISMO .
0.13 ACETALDEIDO .
0.13 LINGUA .
6 0.36 LILACS Generation of reduced nicotinamide adenine dinucleotide phosphate mediated by glucose-6-phosphate dehydrogenase and 6-phosphogluconate dehydrogenase in neutrophils of thoroughbred horses^ien . 
[Em vinte cavalos Puro-Sangue Inglês determinou-se a atividade da glicose-6-fosfato-desidrogenase e da 6-fosfogliconato-desidrogenase de neutrófilos, encontrando-se atividades específicas de 945 ñ 288 mUI mg-1 de proteína e 375 ñ 88 mUI mg-1 de proteína, respectivamente, por minuto a 37§C^ipt] + Generation of reduced nicotinamide adenine dinucleotide phosphate mediated by glucose-6-phosphate dehydrogenase and 6-phosphogluconate dehydrogenase in neutrophils of thoroughbred horses^ien; Braz j vet res anim sci .
Em vinte cavalos Puro-Sangue Inglês determinou-se a atividade da glicose-6-fosfato-desidrogenase e da 6-fosfogliconato-desidrogenase de neutrófilos, encontrando-se atividades específicas de 945 ñ 288 mUI mg-1 de proteína e 375 ñ 88 mUI mg-1 de proteína, respectivamente, por minuto a 37§C^ipt .

  0.25 DEFICIENCIA DE GLUCOSEFOSFATO DESIDROGENASE . 
0.21 /SANGUE .
0.19 /ENZIMOLOGIA .
0.18 GLUCOSEFOSFATO DESIDROGENASE .
0.18 /METABOLISMO .
7 0.35 LILACS Hyperandrogenism due to 3 beta-hydroxysteroid dehydrogenase deficiency with accessory adrenocortical tissue: a hormonal and metabolic evaluation^ien . 
[1 Adrenal ectopic tissue has been detected in the paragonadal region of normal women In patients with congenital adrenal hyperplasia due to 21-hydroxylase (21-OH) deficiency, the manifestation of hyperplasia of paragonadal accessory adrenal tissue has been usually reported to occur in males Probably, this is the first report of a female with 3 beta-hydroxysteroid dehydrogenase (3 beta-HSD) deficie .
1 Adrenal ectopic tissue has been detected in the paragonadal region of normal women In patients with congenital adrenal hyperplasia due to 21-hydroxylase (21-OH) deficiency, the manifestation of hyperplasia of paragonadal accessory adrenal tissue has been usually reported to occur in males Probably, this is the first report of a female with 3 beta-hydroxysteroid dehydrogenase (3 beta-HSD) deficie .. lower than that of 14 normal women (area under the curve, 3 beta-HSD = 17,680 vs 50,034 pmol/l for the control group over a period of 3 h after glucose ingestion)(ABSTRACT TRUNCATED AT 250 WORDS)^ien .

  0.24 /DEFICIENCIA . 
0.22 3-HIDROXIESTEROIDE DESIDROGENASES .
0.19 /GENETICA .
0.17 /DIAGNOSTICO .
0.17 HIPERPLASIA SUPRARRENAL CONGENITA .
8 0.35 LILACS Effect of the ionic environment on the activity of the mitochondrial L-malate dehydrogenase^ien . 

  0.25 ASPARAGINA . 
0.25 GLUTAMATO DESIDROGENASE .
0.17 L-LACTATO DESIDROGENASE .
0.16 MALATO DESIDROGENASE .
0.16 CONCENTRACAO OSMOLAR .
9 0.35 LILACS The glyceraldehyde-3-phosphate dehydrogenase gene of Moniliophthora perniciosa, the causal agent of witches' broom disease of Theobroma cacao^ien . 
APDH gene by real-time PCR showed that this gene was more expressed (~13X) in the saprotrophic stage of this hemibiotrophic plant pathogen than in the biotrophic stage when grown in cacao extracts^ien] + The glyceraldehyde-3-phosphate dehydrogenase gene of Moniliophthora perniciosa, the causal agent of witches' broom disease of Theobroma cacao^ien; Genet mol biol .
This report describes the cloning, sequence and expression analysis of the glyceraldehyde-3-phosphate dehydrogenase (GAPDH) gene of Moniliophthora perniciosa, the most important pathogen of cocoa in Brazil Southern blot analysis revealed the presence of a single copy of the GAPDH gene in the M perniciosa genome (MpGAPDH) The complete MpGAPDH coding sequence contained 1,461 bp with eight introns th .. APDH gene by real-time PCR showed that this gene was more expressed (~13X) in the saprotrophic stage of this hemibiotrophic plant pathogen than in the biotrophic stage when grown in cacao extracts^ien .

  0.31 /QUIMICA . 
0.18 PLANTAS .
0.18 GLICOLISE .
0.17 /GENETICA .
0.17 INOSINA MONOFOSFATO .
10 0.35 LILACS Evidence in Colombia of 625G>A polymorphism in the short chain acyl-CoA dehydrogenase gene, a variation which could cause glutaric aciduria in our populations^ien . 
[Introduction: Short-chain acyl-CoA dehydrogenase (SCAD) is a homotetrameric mitochondrial flavoenzyme that catalyzes the initial reaction in short-chain fatty acid b-oxidation The SCAD gene is located on chromosome 12q22 and is approximately 13 kb long with 10 exons and 1236 nucleotides of coding sequence Hereditary SCAD deficiency has been reported and only a few cases of this disorder have been .
Introduction: Short-chain acyl-CoA dehydrogenase (SCAD) is a homotetrameric mitochondrial flavoenzyme that catalyzes the initial reaction in short-chain fatty acid b-oxidation The SCAD gene is located on chromosome 12q22 and is approximately 13 kb long with 10 exons and 1236 nucleotides of coding sequence Hereditary SCAD deficiency has been reported and only a few cases of this disorder have been .. he short-chain acyl-CoA dehydrogenase gene in Colombia, meaning that some people in our populations can be at risk of suffering SCAD deficiency and its main complication: the ethylmalonic aciduria^ien .

  0.28 /GENETICA . 
0.22 POLIMORFISMO GENETICO .
0.17 OXIDACAO .
0.17 /EFEITOS ADVERSOS .
0.17 ACIL-COA DESIDROGENASE .